Abstract
The biochemical mechanisms subserving the inhibitory actions of prostaglandin F2α on ovarian cells are not known. Since the protein kinase C pathway is coupled to steroidogenesis in an inhibitory fashion in pig granulosa cells, we have tested the hypothesis that prostaglandin F2α activates this phospholipid-dependent, calcium-stimulated effector pathway. Using monolayer cultures of swine granulosa cells, we now report that prostaglandin F2α is capable of activating critical components of the protein kinase C pathway, including the production of water-soluble inositol phosphates, liberation of free arachidonic acid, release of endogenous diacylglycerol, and translocation of cytosolic protein kinase C to the phospholipid-enriched membrane microenvironment.
| Original language | English (US) |
|---|---|
| Pages (from-to) | 112-117 |
| Number of pages | 6 |
| Journal | Biochemical and Biophysical Research Communications |
| Volume | 149 |
| Issue number | 1 |
| DOIs | |
| State | Published - Nov 30 1987 |
ASJC Scopus subject areas
- Biophysics
- Biochemistry
- Molecular Biology
- Cell Biology
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