Antiepileptic drugs: How they work in headache

Keyphrases

• Amino 16%
• Antiepileptic Drugs 100%
• Biochemical Features 16%
• Calcium Ion Channel 66%
• Clinical Features 16%
• Epilepsy 33%
• GABA Metabolism 16%
• GABAA Receptor (GABAAR) 16%
• Gabapentin 66%
• Glutamate 16%
• Glutamate Receptors 16%
• Head Pain 16%
• Headache 100%
• Ion Channel Activity 16%
• Kainate 16%
• L-type Calcium 33%
• Mechanism of Action 16%
• Migraine 66%
• Migraine Model 16%
• Migraine Prevention 16%
• N-methyl-D-aspartate 16%
• Neurotransmission 16%
• Nociception 16%
• Nociceptive Pathways 16%
• Pathophysiology 33%
• Pharmacologic Agents 16%
• Propionic Acid 16%
• Sodium Channel 16%
• Succinate 16%
• T-type Calcium 16%
• Topiramate 83%
• Valproate 66%
• Voltage-dependent Activation 16%
• γ-aminobutyric Acid 16%

Pharmacology, Toxicology and Pharmaceutical Science

• 4 Aminobutyric Acid 83%
• 4 Aminobutyric Acid Receptor 16%
• Alpha Amino 3 Hydroxy 5 Methyl 4 Isoxazolepropionic Acid 16%
• Anticonvulsive Agent 100%
• Aspartic Acid 16%
• Calcium Channel 66%
• Clinical Feature 16%
• Gabapentin 66%
• Glutamate Receptor 16%
• Glutamic Acid 16%
• Headache 100%
• Ion Channel 16%
• Kainic Acid 16%
• Migraine 100%
• Pathophysiology 33%
• Sodium Ion 16%
• Sodium Valproate 66%
• Succinic Acid 16%
• Topiramate 83%

Neuroscience

• AMPA 16%
• Anticonvulsant 100%
• Aspartic Acid 16%
• Auras 16%
• Calcium Channel 66%
• GABA Receptor 16%
• Gabapentin 66%
• Gamma-Aminobutyric Acid 83%
• Glutamate Receptor 16%
• Glutamic Acid 16%
• Headache 100%
• Ion Channel 16%
• Metabolic Pathway 16%
• Migraine 100%
• Nociception 16%
• Succinate 16%
• Synaptic Transmission 16%
• Topiramate 83%